Search results for "NK Cell Lectin-Like Receptor Subfamily B"

showing 3 items of 3 documents

A novel epitope of N-CAM defines precursors of human adherent NK cells

2004

AbstractActivated, adherent natural killer (A-NK) cells represent a distinct subpopulation of interleukin (IL)-2-stimulated NK cells, which are selectively endowed with the increased expression of integrins and ability to adhere to solid surfaces, migrate into, infiltrate, and destroy cancerous tissues. The present study defines the phenotype and functions of precursors of A-NK (pre-A-NK) cells in humans. Peripheral blood pre-A-NK cells, in contrast to the rest of NK cells, express a novel epitope of CD56 neuronal cell adhesion molecule, termed ANK-1, and increased cell-surface levels of integrins. Pre-A-NK cells also express low levels of CD56 and CD161, and some express CD162 receptor, do…

Cytotoxicity ImmunologicIntegrinsLymphoid TissueImmunologyCell CountBiologyCD49bImmunophenotypingEpitopesInterleukin 21NK-92Cell AdhesionHumansImmunology and AllergyCell LineageLectins C-TypeAntigen-presenting cellCells CulturedCell ProliferationMembrane GlycoproteinsLymphokine-activated killer cellStem CellsJanus kinase 3Cell MembraneReceptors IgGAntibodies MonoclonalCell DifferentiationCell BiologyNatural killer T cellCD56 AntigenCell biologyKiller Cells NaturalChemotaxis LeukocyteAntigens SurfaceInterleukin 12Interleukin-2NK Cell Lectin-Like Receptor Subfamily BJournal of Leukocyte Biology
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Resistance of natural killer T cell-deficient mice to systemic Shwartzman reaction.

2000

The generalized Shwartzman reaction in mice which had been primed and challenged with lipopolysaccharide (LPS) depends on interleukin (IL)-12-induced interferon (IFN)-gamma production at the priming stage. We examined the involvement in the priming mechanism of the unique population of Valpha14 natural killer T (NKT) cells because they promptly produce IFN-gamma after IL-12 stimulation. We report here that LPS- or IL-12-primed NKT cell genetically deficient mice were found to be resistant to LPS-elicited mortality. This outcome can be attributed to the reduction of IFN-gamma production, because injection of recombinant mouse IFN-gamma, but not injection of IL-12, effectively primed the NKT …

LipopolysaccharidesShwartzman phenomenonReceptors Antigen T-Cell alpha-betaImmunologyPopulationPriming (immunology)Mice SCIDBiologyLymphocyte DepletionInterferon-gammaMiceInterferonmedicineImmunology and AllergyAnimalsInterferon gammaLectins C-TypeAntigenseducationeducation.field_of_studyMice Inbred BALB Cinterferon γTumor Necrosis Factor-alphalipopolysaccharideBrief Definitive ReportInterleukinProteinsShwartzman reactionNatural killer T cellmedicine.diseaseInterleukin-12Immunity Innatenatural killer T cellsKiller Cells NaturalMice Inbred C57BLImmunologyAntigens SurfaceInterleukin 12interleukin 12medicine.drugNK Cell Lectin-Like Receptor Subfamily BShwartzman PhenomenonThe Journal of experimental medicine
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Th1 and Th17 lymphocytes expressing CD161 are implicated in giant cell arteritis and polymyalgia rheumatica pathogenesis.

2012

International audience; OBJECTIVE: Giant cell arteritis (GCA) is the most frequently occurring vasculitis in elderly individuals, and its pathogenesis is not fully understood. The objective of this study was to decipher the role of the major CD4+ T cell subsets in GCA and its rheumatologic form, polymyalgia rheumatica (PMR). METHODS: A prospective study of the phenotype and the function of major CD4+ T cell subsets (Th1, Th17, and Treg cells) was performed in 34 untreated patients with GCA or PMR, in comparison with 31 healthy control subjects and with the 27 treated patients who remained after the 7 others withdrew. RESULTS: Compared with control subjects, patients with GCA and patients wi…

MalePathologyMESH: Th17 CellsCellMESH : AgedMESH : Prospective StudiesMESH: Flow CytometryT-Lymphocytes RegulatoryPathogenesisMESH : T-Lymphocytes Regulatory0302 clinical medicineimmune system diseasesMESH : Th1 CellsImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyPharmacology (medical)MESH : FemaleProspective Studiesskin and connective tissue diseasesCells CulturedMESH: Aged0303 health sciencesMESH: Middle Agedmedicine.diagnostic_testMESH: Giant Cell ArteritisCell DifferentiationMESH : AdultMiddle AgedFlow CytometryMESH : NK Cell Lectin-Like Receptor Subfamily B3. Good healthMESH: NK Cell Lectin-Like Receptor Subfamily Bmedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyFemaleVasculitisMESH : Cell DifferentiationGlucocorticoidmedicine.drugNK Cell Lectin-Like Receptor Subfamily BMESH: Cells CulturedAdultMESH: Cell Differentiationmedicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyMESH : Flow CytometryT cellMESH : MaleImmunologyGiant Cell ArteritisBiologyPolymyalgia rheumatica03 medical and health sciencesRheumatologyBiopsyMESH : Cells CulturedmedicineMESH : Th17 CellsHumansMESH : Middle Aged030304 developmental biologyAged030203 arthritis & rheumatologyMESH: HumansMESH: T-Lymphocytes RegulatoryMESH : HumansMESH: AdultTh1 Cellsmedicine.diseaseMESH : Giant Cell ArteritisMESH: Prospective StudiesMESH: MaleGiant cell arteritisMESH: Th1 CellsPolymyalgia RheumaticaMESH: Polymyalgia RheumaticaImmunologyTh17 CellsMESH : Polymyalgia RheumaticaMESH: Female
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